Anabell Juárez-Martínez
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Anabell Juárez-Martínez has a total of 5 co-authors. Here are the top co-authors by number of shared publications:
- Ahgiel Jiménez-Ruiz
Intensive Care Unit, Hospital General la Perla, Mexico City 57830, Mexico
1 shared publication - Daphne Marisol Montes-Ventura
Department of Toxicology, Hospital Juárez de México, Mexico City 07760, Mexico
1 shared publication - Jesús del Carmen Madrigal-Anaya
Department of Toxicology, Hospital Juárez de México, Mexico City 07760, Mexico
1 shared publication - Ramsés Dorado-García
Department of Toxicology, Hospital Juárez de México, Mexico City 07760, Mexico
1 shared publication - Yessika Paola Rodríguez-Torres
Encargada del Servicio de Toxicología Clínica y profesor adjunto de la especialidad
1 shared publication
Latest Publications
- Open Access
Zinc Phosphide Poisoning: From A to Z
Zinc phosphide is a rodenticide that is used in agricultural, urban and industrial environments in México. After ingestion, it reacts with hydrochloric acid, hydrolyzing into phosphine. It causes cellular hypoxia via mitochondrial toxicity, resulting in multiple organ dysfunction and death. There is no antidote or specific treatment for zinc phosphide toxicity. We present the case of a 45-year-old female who ingested zinc phosphide with suicidal intent. On arrival at the emergency department, she had multisystemic disorders. Supportive care, decontamination and antidotal therapy were initiated. Subsequently, she evolved to clinical improvement with a resolution of the biochemical abnormalities of tissue hypoperfusion. She was discharged on day 7 without complications. In this review, we provide updated therapeutic options and discuss their specific pathophysiological basis.
Open AccessZinc Phosphide Poisoning: From A to Z
Zinc phosphide is a rodenticide that is used in agricultural, urban and industrial environments in México. After ingestion, it reacts with hydrochloric acid, hydrolyzing into phosphine. It causes cellular hypoxia via mitochondrial toxicity, resulting in multiple organ dysfunction and death. There is no antidote or specific treatment for zinc phosphide toxicity. We present the case of a 45-year-old female who ingested zinc phosphide with suicidal intent. On arrival at the emergency department, she had multisystemic disorders. Supportive care, decontamination and antidotal therapy were initiated. Subsequently, she evolved to clinical improvement with a resolution of the biochemical abnormalities of tissue hypoperfusion. She was discharged on day 7 without complications. In this review, we provide updated therapeutic options and discuss their specific pathophysiological basis.