Ruud van der Breggen
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Ruud van der Breggen has a total of 60 co-authors. Here are the top co-authors by number of shared publications:
- Marieke E. Ijsselsteijn
Department of Pathology, Leiden University Medical Center, Leiden, The Netherlands
46 shared publications - Noel F. C. C. de Miranda
Department of Pathology, Leiden University Medical Center, Leiden, The Netherlands
45 shared publications - Manon van der Ploeg
Department of Pathology, Leiden University Medical Center, Leiden, The Netherlands
44 shared publications - Dina Ruano
Department of Pathology, Leiden University Medical Center
41 shared publications - Elisabeth M. E. Verdegaal
Department of Medical Oncology, Oncode Institute, Leiden University Medical Center, Leiden, The Netherlands
41 shared publications - Jitske van Den Bulk
Department of Pathology, Leiden University Medical Center, Leiden, The Netherlands
41 shared publications
Latest Publications
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S6 from Neoantigen Targetability in Progressive Advanced Melanoma
Expression of tumor associated antigens in early and later metastatic lesions.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S6 from Neoantigen Targetability in Progressive Advanced Melanoma
Expression of tumor associated antigens in early and later metastatic lesions.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S3 from Neoantigen Targetability in Progressive Advanced Melanoma
HLA class I genotypes as determined by exome and transcriptome sequencing.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S3 from Neoantigen Targetability in Progressive Advanced Melanoma
HLA class I genotypes as determined by exome and transcriptome sequencing.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from Neoantigen Targetability in Progressive Advanced Melanoma
Whole-exome sequencing strategy.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from Neoantigen Targetability in Progressive Advanced Melanoma
Whole-exome sequencing strategy.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S2 from Neoantigen Targetability in Progressive Advanced Melanoma
HLA class I and II expression on melanoma cell lines derived from early and later metastatic lesions.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S2 from Neoantigen Targetability in Progressive Advanced Melanoma
HLA class I and II expression on melanoma cell lines derived from early and later metastatic lesions.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Methods S1 from Neoantigen Targetability in Progressive Advanced Melanoma
More detailed information on: 1) Immune-fluorescent staining, 2) Mass spectrometry, 3) MLTC and TIL cultures.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Methods S1 from Neoantigen Targetability in Progressive Advanced Melanoma
More detailed information on: 1) Immune-fluorescent staining, 2) Mass spectrometry, 3) MLTC and TIL cultures.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S5 from Neoantigen Targetability in Progressive Advanced Melanoma
Putative neoantigens in early and later metastatic lesions.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S5 from Neoantigen Targetability in Progressive Advanced Melanoma
Putative neoantigens in early and later metastatic lesions.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S5 from Neoantigen Targetability in Progressive Advanced Melanoma
Eluted tumor-associated antigen-derived peptides.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S5 from Neoantigen Targetability in Progressive Advanced Melanoma
Eluted tumor-associated antigen-derived peptides.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S1 from Neoantigen Targetability in Progressive Advanced Melanoma
Immunohistochemical staining of proteins involved in T cell recognition and/or inhibition.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S1 from Neoantigen Targetability in Progressive Advanced Melanoma
Immunohistochemical staining of proteins involved in T cell recognition and/or inhibition.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S3 from Neoantigen Targetability in Progressive Advanced Melanoma
T cell recognition of melanoma cell lines derived from early and later metastatic lesions.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S3 from Neoantigen Targetability in Progressive Advanced Melanoma
T cell recognition of melanoma cell lines derived from early and later metastatic lesions.
- Open Access
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S4 from Neoantigen Targetability in Progressive Advanced Melanoma
HLA class II expression and recognition of early and later metastatic lesion- derived tumor cells from patient Mel5 by CD4+ and CD8⁺ T cells.
- 25 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S4 from Neoantigen Targetability in Progressive Advanced Melanoma
HLA class II expression and recognition of early and later metastatic lesion- derived tumor cells from patient Mel5 by CD4+ and CD8⁺ T cells.