J. M. Hougard
Research Subjects
Research Interests
J. M. Hougard has a total of 10 co-authors. Here are the top co-authors by number of shared publications:
- Abdoulaye Diabaté
Institut de Recherche en Sciences de la Santé (IRSS), Bobo-Dioulasso 01, Burkina Faso
1 shared publication - Cécile Brengues
Institut de Recherche pour le Développement, UR016, B.P. 64501, 34397, Montpellier, France
1 shared publication - Fabrice Chandre
Institut de Recherche pour le Développement (IRD), UMR MIVEGEC (UM1-UM2-CNRS 5290-IRD 224) Maladies Infectieuses et Vecteurs, Ecologie, Génétique, Evolution et Contrôle, Laboratoire de Lutte contre les Insectes Nuisibles (LIN), Montpellier, France
1 shared publication - Frédéric Simard
MIVEGEC, University of Montpellier, IRD, CNRS, Montpellier, France
1 shared publication - Janet Hemingway
Liverpool School of Tropical Medicine, Pembroke Place, Liverpool L3 5QA, UK
1 shared publication - Martin Akogbeto
Centre de Recherche Entomologique de Cotonou, 06 BP 2604, Benin
1 shared publication
Latest Publications
- Open Access
- 9 December 2004
- Published by Wiley in Tropical Medicine & International Health
- Vol. 9 (12) , 1267-1273
The spread of the Leu‐Phe kdr mutation through Anopheles gambiae complex in Burkina Faso: genetic introgression and de novo phenomena
Summary: During extensive sampling in Burkina Faso and other African countries, the Leu‐Phe mutation producing the kdr pyrethroid resistance phenotype was reported in both Anopheles gambiae ss and A. arabiensis. This mutation was widely distributed at high frequency in the molecular S form of A. gambiae while it has been observed at a very low frequency in both the molecular M form and A. arabiensis in Burkina Faso. While the mutation in the M form is inherited through an introgression from the S form, its occurrence is a new and independent mutation event in A. arabiensis. Three nucleotides in the upstream intron of the kdr mutation differentiated A. arabiensis from A. gambiae ss and these specific nucleotides were associated with kdr mutation in A. arabiensis. Ecological divergences which facilitated the spread of the kdr mutation within the complex of A. gambiae ss in West Africa, are discussed.
- 9 December 2004
- Published by Wiley in Tropical Medicine & International Health
- Vol. 9 (12) , 1267-1273
Open AccessThe spread of the Leu‐Phe kdr mutation through Anopheles gambiae complex in Burkina Faso: genetic introgression and de novo phenomena
Summary: During extensive sampling in Burkina Faso and other African countries, the Leu‐Phe mutation producing the kdr pyrethroid resistance phenotype was reported in both Anopheles gambiae ss and A. arabiensis. This mutation was widely distributed at high frequency in the molecular S form of A. gambiae while it has been observed at a very low frequency in both the molecular M form and A. arabiensis in Burkina Faso. While the mutation in the M form is inherited through an introgression from the S form, its occurrence is a new and independent mutation event in A. arabiensis. Three nucleotides in the upstream intron of the kdr mutation differentiated A. arabiensis from A. gambiae ss and these specific nucleotides were associated with kdr mutation in A. arabiensis. Ecological divergences which facilitated the spread of the kdr mutation within the complex of A. gambiae ss in West Africa, are discussed.