Emilia Mazur
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Emilia Mazur has a total of 3 co-authors. Here are the top co-authors by number of shared publications:
- Frank Edlich
Institute of Biochemistry, Faculty of Veterinary Medicine, University of Leipzig, Leipzig, Germany
1 shared publication - Frederike Werry
Institute of Biochemistry, Faculty of Veterinary Medicine, University of Leipzig, 04103 Leipzig, Germany
1 shared publication - Lars F. H. Theyse
Department for Small Animals, Soft Tissue and Orthopaedic Surgery Service, College of Veterinary Medicine, University of Leipzig, Leipzig, Germany
1 shared publication
Latest Publications
- Open Access
- 22 August 2023
- Published by MDPI AG in International Journal of Molecular Sciences
- Vol. 24 (17) , 13028
Apoptosis Regulation in Osteoarthritis and the Influence of Lipid Interactions
Osteoarthritis (OA) is one of the most common chronic diseases in human and animal joints. The joints undergo several morphological and histological changes during the development of radiographically visible osteoarthritis. The most discussed changes include synovial inflammation, the massive destruction of articular cartilage and ongoing joint destruction accompanied by massive joint pain in the later stadium. Either the increased apoptosis of chondrocytes or the insufficient apoptosis of inflammatory macrophages and synovial fibroblasts are likely to underly this process. In this review, we discuss the current state of research on the pathogenesis of OA with special regard to the involvement of apoptosis.
- 22 August 2023
- Published by MDPI AG in International Journal of Molecular Sciences
- Vol. 24 (17) , 13028
Open AccessApoptosis Regulation in Osteoarthritis and the Influence of Lipid Interactions
Osteoarthritis (OA) is one of the most common chronic diseases in human and animal joints. The joints undergo several morphological and histological changes during the development of radiographically visible osteoarthritis. The most discussed changes include synovial inflammation, the massive destruction of articular cartilage and ongoing joint destruction accompanied by massive joint pain in the later stadium. Either the increased apoptosis of chondrocytes or the insufficient apoptosis of inflammatory macrophages and synovial fibroblasts are likely to underly this process. In this review, we discuss the current state of research on the pathogenesis of OA with special regard to the involvement of apoptosis.