CWChangyue Wu
Changyue Wu
*This is an algorithmically generated author record based on publicly available information.
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Changyue Wu has a total of 18 co-authors. Here are the top co-authors by number of shared publications:
- Canbin Dong
Department of Dermatology, Huashan Hospital, Fudan University, Shanghai, China
1 shared publication - Dan Wu
Department of Dermatology, Huashan Hospital, Fudan University, Shanghai, China
1 shared publication - Gongyun Lei
Department of Pathophysiology, School of Medicine, Nantong University, Jiangsu, China
1 shared publication - Hua Xian
Department of Pediatric Surgery, Affiliated Hospital of Nantong University, Nantong University, Jiangsu, China
1 shared publication - Jianlan Liu
Department of Dermatology, Huashan Hospital, Fudan University, Shanghai, China
1 shared publication - Jie Ren
Department of Dermatology, Huashan Hospital, Fudan University, Shanghai, China
1 shared publication
Latest Publications
- Open Access
- 17 June 2026
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 146 (11) , 3162-3173.e10
X-ray irradiation suppresses keloid growth by inducing G2/M arrest through the phytosphingosine–KIF20A axis
- 17 June 2026
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 146 (11) , 3162-3173.e10
Open AccessX-ray irradiation suppresses keloid growth by inducing G2/M arrest through the phytosphingosine–KIF20A axis
- 11 October 2023
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 144 (3) , 645-658.e7
Apolipoprotein A-I Binding Protein Inhibits the Formation of Infantile Hemangioma through Cholesterol-Regulated Hypoxia-Inducible Factor 1α Activation
- 11 October 2023
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 144 (3) , 645-658.e7
Apolipoprotein A-I Binding Protein Inhibits the Formation of Infantile Hemangioma through Cholesterol-Regulated Hypoxia-Inducible Factor 1α Activation