Mina Hibino
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Mina Hibino has a total of 18 co-authors. Here are the top co-authors by number of shared publications:
- Paul Thevenot
Institute of Translational Research, Ochsner Health System, New Orleans, LA 70121, USA
35 shared publications - Ari Cohen
Multi-Organ Transplant Institute, Ochsner Health System, New Orleans, LA 70121, USA
34 shared publications - Kelley Núñez
Institute of Translational Research, Ochsner Health System, New Orleans, LA 70121, USA
34 shared publications - Tyler Sandow
Interventional Radiology, Ochsner Health System, New Orleans, LA 70121, USA
34 shared publications - Juan Gimenez
Department of Radiology, Ochsner Medical Center, New Orleans, LA USA
32 shared publications - Daniel Fort
Affiliation Center for Outcomes Research, Ochsner Health, New Orleans, Louisiana, United States of America
3 shared publications
Latest Publications
- Open Access
- 26 March 2025
- Published by American Association for Cancer Research (AACR)
Figure 1 from Yttrium-90 Induces an Effector Memory Response with Neoantigen Clonotype Expansion: Implications for Immunotherapy
90Y-TARE induced changes in CD4+ T-cell populations in early–intermediate stage HCC. A, Percentage of CD4+ T cells of total T cells remained stable post-90Y, whereas (B) the percentage of naïve CD4+ increased following 90Y-TARE. Decreased percentage of (C) memory stem CD4+ T cells and (D) central memory CD4+ T cells was observed following 90Y-TARE. E, Increase in the percentage of effector memory/effector CD4+ T cells, whereas senescent T cells with (F) CD57+ and (G) KLRG1+ remained unchanged post-90Y. An increase in the median fluorescence intensity for the exhaustion marker (H) CTLA4 was observed, whereas (I) LAG3 and (J) PD1 remained stable after 90Y. Blue and red boxes represent IQR, and the solid horizontal line represents median. MFI, median fluorescence intensity; KLGR1, killer lectin–like receptor G1.
- 26 March 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure 1 from Yttrium-90 Induces an Effector Memory Response with Neoantigen Clonotype Expansion: Implications for Immunotherapy
90Y-TARE induced changes in CD4+ T-cell populations in early–intermediate stage HCC. A, Percentage of CD4+ T cells of total T cells remained stable post-90Y, whereas (B) the percentage of naïve CD4+ increased following 90Y-TARE. Decreased percentage of (C) memory stem CD4+ T cells and (D) central memory CD4+ T cells was observed following 90Y-TARE. E, Increase in the percentage of effector memory/effector CD4+ T cells, whereas senescent T cells with (F) CD57+ and (G) KLRG1+ remained unchanged post-90Y. An increase in the median fluorescence intensity for the exhaustion marker (H) CTLA4 was observed, whereas (I) LAG3 and (J) PD1 remained stable after 90Y. Blue and red boxes represent IQR, and the solid horizontal line represents median. MFI, median fluorescence intensity; KLGR1, killer lectin–like receptor G1.