Brian Rabinovich
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Brian Rabinovich has a total of 32 co-authors. Here are the top co-authors by number of shared publications:
- Amit M. Deshpande
Mythic Therapeutics Inc., Waltham, Massachusetts
33 shared publications - Chunxiao Xu
EMD Serono Res Ctr, Res Unit Oncol, 45 Middlesex Turnpike, Billerica, MA 01821 USA
33 shared publications - Laura Helming
Research Unit Oncology, EMD Serono, EMD Serono Research & Development Institute
33 shared publications - Lindsay Webb
Research Unit Oncology, EMD Serono, Billerica, Massachusetts
33 shared publications - Marat Alimzhanov
Research Unit Oncology, EMD Serono, Billerica, Massachusetts
33 shared publications - Maria Soloviev
Discovery and Development Technologies, EMD Serono, Billerica, Massachusetts
33 shared publications
Latest Publications
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S1 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
M9567 mAb2 TM structure and molecules used for primary research
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
M9567 mAb2 TM structure and molecules used for primary research
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S13 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
Tumor-targeted immune activation does not result in increased liver inflammation
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S13 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
Tumor-targeted immune activation does not result in increased liver inflammation
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S2 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
M9657 is not appreciably internalized in MSLN+ cells
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
M9657 is not appreciably internalized in MSLN+ cells
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S10 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m generates dose-dependent antitumor efficacy and long-term tumor antigen-specific immune protection.
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S10 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m generates dose-dependent antitumor efficacy and long-term tumor antigen-specific immune protection.
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S7 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m characterization on cellular binding, in vitro agonism and in vivo distribution
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S7 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m characterization on cellular binding, in vitro agonism and in vivo distribution
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S8 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m surrogate and M9657 chimeric antibodies display significant antitumor efficacy in syngeneic models
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S8 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m surrogate and M9657 chimeric antibodies display significant antitumor efficacy in syngeneic models
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S8 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m surrogate and M9657 chimeric antibodies display significant antitumor efficacy in syngeneic models
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S8 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m surrogate and M9657 chimeric antibodies display significant antitumor efficacy in syngeneic models
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S2 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
M9657 is not appreciably internalized in MSLN+ cells
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
M9657 is not appreciably internalized in MSLN+ cells
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S11 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m increases infiltration of CD8+ T cells but not other immune subsets in the TME and spleen of the CT26-KSA tumor model
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S11 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
FS122m increases infiltration of CD8+ T cells but not other immune subsets in the TME and spleen of the CT26-KSA tumor model
- Open Access
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Figure S9 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
MuMSLN was depleted in the EMT‑6‑ MSLN‑del cell line
- 2 February 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S9 from M9657 Is a Bispecific Tumor-Targeted Anti-CD137 Agonist That Induces MSLN-Dependent Antitumor Immunity without Liver Inflammation
MuMSLN was depleted in the EMT‑6‑ MSLN‑del cell line