Erika Schirinzi
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Erika Schirinzi has a total of 4 co-authors. Here are the top co-authors by number of shared publications:
- Francesca Torri
Department of Clinical and Experimental Medicine, University of Pisa, 56126 Pisa, Italy
1 shared publication - Gabriele Siciliano
Department of Clinical and Experimental Medicine, Neurology Unit, University of Pisa, Pisa, Italy
1 shared publication - Giulia Ricci
Department of Clinical and Experimental Medicine, University of Pisa, Pisa, Italy
1 shared publication - Michelangelo Mancuso
Department of Clinical and Experimental Medicine, Neurological Institute, University of Pisa, 56126 Pisa, Italy
1 shared publication
Latest Publications
- Open Access
- 30 December 2023
- Published by MDPI AG in Biomolecules
- Vol. 14 (1) , 50
Biomolecules of Muscle Fatigue in Metabolic Myopathies
Metabolic myopathies are a group of genetic disorders that affect the normal functioning of muscles due to abnormalities in metabolic pathways. These conditions result in impaired energy production and utilization within muscle cells, leading to limitations in muscle function with concomitant occurrence of related signs and symptoms, among which fatigue is one of the most frequently reported. Understanding the underlying molecular mechanisms of muscle fatigue in these conditions is challenging for the development of an effective diagnostic and prognostic approach to test targeted therapeutic interventions. This paper outlines the key biomolecules involved in muscle fatigue in metabolic myopathies, including energy substrates, enzymes, ion channels, and signaling molecules. Potential future research directions in this field are also discussed.
- 30 December 2023
- Published by MDPI AG in Biomolecules
- Vol. 14 (1) , 50
Open AccessBiomolecules of Muscle Fatigue in Metabolic Myopathies
Metabolic myopathies are a group of genetic disorders that affect the normal functioning of muscles due to abnormalities in metabolic pathways. These conditions result in impaired energy production and utilization within muscle cells, leading to limitations in muscle function with concomitant occurrence of related signs and symptoms, among which fatigue is one of the most frequently reported. Understanding the underlying molecular mechanisms of muscle fatigue in these conditions is challenging for the development of an effective diagnostic and prognostic approach to test targeted therapeutic interventions. This paper outlines the key biomolecules involved in muscle fatigue in metabolic myopathies, including energy substrates, enzymes, ion channels, and signaling molecules. Potential future research directions in this field are also discussed.