Tie Li
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Tie Li has a total of 28 co-authors. Here are the top co-authors by number of shared publications:
- Albert Lai
Department of Neurology, David Geffen School of Medicine, University of California, Los Angeles , Los Angeles, CA
118 shared publications - David A. Nathanson
Department of Molecular and Medical Pharmacology, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA, USA
87 shared publications - Linda M. Liau
Department of Neurosurgery, David Geffen School of Medicine, University of California, Los Angeles, California, Los Angeles, CA, USA
87 shared publications - Terry J. Prins
Department of Neurology, UCLA Medical Center, Los Angeles, California
87 shared publications - Harley I. Kornblum
Department of Psychiatry and Behavioral Sciences and the UCLA Intellectual and Developmental Disabilities Research Center, David Geffen School of Medicine, UCLA, Los Angeles, USA
62 shared publications - Janet Treger
Department of Neurosurgery, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California. 1
56 shared publications
Latest Publications
- Open Access
- 28 March 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data 1 from Epigenetic Induction of Cancer-Testis Antigens and Endogenous Retroviruses at Single-Cell Level Enhances Immune Recognition and Response in Glioma
Isoplexis fluorescence values per cytokine per cell
- 28 March 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 1 from Epigenetic Induction of Cancer-Testis Antigens and Endogenous Retroviruses at Single-Cell Level Enhances Immune Recognition and Response in Glioma
Isoplexis fluorescence values per cytokine per cell
- Open Access
- 28 March 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data 2 from Epigenetic Induction of Cancer-Testis Antigens and Endogenous Retroviruses at Single-Cell Level Enhances Immune Recognition and Response in Glioma
HERV raw counts and FPKM values
- 28 March 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 2 from Epigenetic Induction of Cancer-Testis Antigens and Endogenous Retroviruses at Single-Cell Level Enhances Immune Recognition and Response in Glioma
HERV raw counts and FPKM values
- Open Access
- 28 March 2025
- Published by American Association for Cancer Research (AACR)
Figure 5 from Epigenetic Induction of Cancer-Testis Antigens and Endogenous Retroviruses at Single-Cell Level Enhances Immune Recognition and Response in Glioma
CTA induction but not downstream interferon-downstream stimulation by DAC is correlated with CpG demethylation in glioma cells. A, Heatmap and hierarchical clustering of differentially expressed CTA. Scale represents z-score of DESeq2 counts. B, Violin plot of beta value distribution between treatment conditions for CpG probes corresponding to differentially upregulated CTA (P-value = 0.028; Wilcoxon rank sum test with continuity correction). C, Scatter plot of all CpG island probes containing intersectional DNA methylation log2FoldChange and RNA expression log2FoldChange of annotated genes in black (n = 115,801 CpG island probes). CpG island probes corresponding to CTA with Pearson correlation are highlighted in (red; n = 535 CpG probes; n = 65 CTA genes). D, Enricher analysis of DESEQ2 differentially upregulated genes (Hallmark IFNγ P = 0.00548; Hallmark IFNα P = 0.00548; Hallmark inflammatory response P = 0.0426) and downregulated genes (Hallmark mTORC1 signaling P = 1.59e−3; Hallmark mitotic spindle P = 1.59e−3, Hallmark G2M Checkpoint P = 7.77e−5; Hallmark E2F targets P = 7.77e−5). Hallmark MSigDB signature adjusted P values determined using Fisher exact test adjusted by Benjamini–Hochberg correction. E, Violin plot of beta value distribution between treatment conditions for CpG probes corresponding to differentially upregulated DAC_UP_MSigDB_Hallmark genes (P-value = 0.497; Wilcoxon rank sum test with continuity correction). F, Scatter plot of intersectional DNA methylation log2FoldChange and RNA expression log2FoldChange CpG island probes. CpG probes corresponding to the Hallmark Interferon Alpha Response gene set with Pearson correlation are highlighted in blue (n = 471 probes; n = 53 genes).
- 28 March 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure 5 from Epigenetic Induction of Cancer-Testis Antigens and Endogenous Retroviruses at Single-Cell Level Enhances Immune Recognition and Response in Glioma
CTA induction but not downstream interferon-downstream stimulation by DAC is correlated with CpG demethylation in glioma cells. A, Heatmap and hierarchical clustering of differentially expressed CTA. Scale represents z-score of DESeq2 counts. B, Violin plot of beta value distribution between treatment conditions for CpG probes corresponding to differentially upregulated CTA (P-value = 0.028; Wilcoxon rank sum test with continuity correction). C, Scatter plot of all CpG island probes containing intersectional DNA methylation log2FoldChange and RNA expression log2FoldChange of annotated genes in black (n = 115,801 CpG island probes). CpG island probes corresponding to CTA with Pearson correlation are highlighted in (red; n = 535 CpG probes; n = 65 CTA genes). D, Enricher analysis of DESEQ2 differentially upregulated genes (Hallmark IFNγ P = 0.00548; Hallmark IFNα P = 0.00548; Hallmark inflammatory response P = 0.0426) and downregulated genes (Hallmark mTORC1 signaling P = 1.59e−3; Hallmark mitotic spindle P = 1.59e−3, Hallmark G2M Checkpoint P = 7.77e−5; Hallmark E2F targets P = 7.77e−5). Hallmark MSigDB signature adjusted P values determined using Fisher exact test adjusted by Benjamini–Hochberg correction. E, Violin plot of beta value distribution between treatment conditions for CpG probes corresponding to differentially upregulated DAC_UP_MSigDB_Hallmark genes (P-value = 0.497; Wilcoxon rank sum test with continuity correction). F, Scatter plot of intersectional DNA methylation log2FoldChange and RNA expression log2FoldChange CpG island probes. CpG probes corresponding to the Hallmark Interferon Alpha Response gene set with Pearson correlation are highlighted in blue (n = 471 probes; n = 53 genes).