Nan-Chun Wu
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Nan-Chun Wu has a total of 15 co-authors. Here are the top co-authors by number of shared publications:
Chia-Chun Wu
Division of Nephrology, Department of Internal Medicine, Chi Mei Medical Center, Tainan, Taiwan
2 shared publicationsChung-Han Ho
Department of Hospital and Health Care Administration, Chi-Mei Medical Center, Tainan, Taiwan; Department of Pharmacy, Chia Nan University of Pharmacy and Science, Tainan, Taiwan
2 shared publicationsI-Chuang Liao
Department of Pathology, Chi-Mei Medical Center, Tainan, Taiwan
2 shared publicationsJhih-Yuan Shih
School of Medicine and Doctoral Program of Clinical and Experimental Medicine, College of Medicine and Center of Excellence for Metabolic Associated Fatty Liver Disease, National Sun Yat-sen University, Kaohsiung, Taiwan; Division of Cardiology, Department of Internal Medicine, Chi Mei Medical Center, Tainan, Taiwan
2 shared publicationsWei-Chieh Lee
School of Medicine and Doctoral Program of Clinical and Experimental Medicine, College of Medicine and Center of Excellence for Metabolic Associated Fatty Liver Disease, National Sun Yat-sen University, Kaohsiung, Taiwan; Division of Cardiology, Department of Internal Medicine, Chi Mei Medical Center, Tainan, Taiwan
2 shared publications- Wei-Ting Chang
Institute of Clinical Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan
2 shared publications
Latest Publications
- 5 August 2023
- Published by Elsevier in Free Radical Biology and Medicine
- Vol. 208, 103-111
Dapagliflozin protects against doxorubicin-induced nephrotoxicity associated with nitric oxide pathway—A translational study
- 5 August 2023
- Published by Elsevier in Free Radical Biology and Medicine
- Vol. 208, 103-111
Dapagliflozin protects against doxorubicin-induced nephrotoxicity associated with nitric oxide pathway—A translational study
- Open Access
- 1 December 2017
- Published by Wolters Kluwer Health in Medicine
- Vol. 96 (50) , e8949
Purulent constrictive pericarditis caused by Salmonella enteritidis in a patient with adult-onset Still's disease
Purulent pericarditis is a rare and usually fatal disease. Immunodeficiency state and preexisting pericardial effusion can predispose patients to infections. However, we are not aware of similar cases in patients with adult-onset Still's disease (AOSD). In addition, it is seldom caused by Salmonella bacteria. We report a 30-year-old woman with dyspnea on exertion and epigastric fullness. She was newly diagnosed with AOSD 4 months previously and medicated with prednisolone. Transthoracic echocardiography (TTE) and computed tomography revealed a thickened pericardium with loculations in the pericardial space, consistent with purulent constrictive pericarditis. Subsequent cultures of blood and pericardial fluid yielded S enteritidis. She underwent subtotal pericardiectomy through a limited median sternotomy, and antibiotic therapy (ceftriaxone) for 1 month. The New York Heart Association functional classification downgraded from class III to class I. There was no recurrence during the 1-year follow-up. This case presents an opportunity to highlight the importance of considering purulent pericarditis in patients previously diagnosed with AOSD. High clinical suspicion, early diagnosis, and prompt management can result in a better outcome in purulent pericarditis.
- 1 December 2017
- Published by Wolters Kluwer Health in Medicine
- Vol. 96 (50) , e8949
Open AccessPurulent constrictive pericarditis caused by Salmonella enteritidis in a patient with adult-onset Still's disease
Purulent pericarditis is a rare and usually fatal disease. Immunodeficiency state and preexisting pericardial effusion can predispose patients to infections. However, we are not aware of similar cases in patients with adult-onset Still's disease (AOSD). In addition, it is seldom caused by Salmonella bacteria. We report a 30-year-old woman with dyspnea on exertion and epigastric fullness. She was newly diagnosed with AOSD 4 months previously and medicated with prednisolone. Transthoracic echocardiography (TTE) and computed tomography revealed a thickened pericardium with loculations in the pericardial space, consistent with purulent constrictive pericarditis. Subsequent cultures of blood and pericardial fluid yielded S enteritidis. She underwent subtotal pericardiectomy through a limited median sternotomy, and antibiotic therapy (ceftriaxone) for 1 month. The New York Heart Association functional classification downgraded from class III to class I. There was no recurrence during the 1-year follow-up. This case presents an opportunity to highlight the importance of considering purulent pericarditis in patients previously diagnosed with AOSD. High clinical suspicion, early diagnosis, and prompt management can result in a better outcome in purulent pericarditis.