Mai Takahashi
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Mai Takahashi has a total of 12 co-authors. Here are the top co-authors by number of shared publications:
- Abdul G. Khan
Sanders Tri-Institutional Therapeutics Discovery Institute 2 , New York, New York
20 shared publications - Akimasa Hayashi
Department of Pathology, Kyorin University School of Medicine, Tokyo, Japan
20 shared publications - Christine Iacobuzio-Donahue
The David M Rubenstein Center for Pancreatic Cancer Research, Memorial Sloan Kettering Cancer Center, New York, NY
20 shared publications - Dennis Hsu
The Ohio State University Comprehensive Cancer Center–Arthur G. James Cancer Hospital and Richard J. Solove Research Institute, Columbus, Ohio, USA
20 shared publications - Elisa de Stanchina
Antitumor Assessment Core, Memorial Sloan Kettering Cancer Center, New York, NY, USA
20 shared publications - Ethan Ravetch
Laboratory of Systems Cancer Biology, The Rockefeller University, New York, New York
20 shared publications
Latest Publications
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S6 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 6 showing AMIGO2 promotes HIF1a nuclear retention via specific HIF1a residues
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S6 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 6 showing AMIGO2 promotes HIF1a nuclear retention via specific HIF1a residues
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S3 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 3 showing NPTX1 is over-expressed in human primary and metastatic PDAC tumors
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S3 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 3 showing NPTX1 is over-expressed in human primary and metastatic PDAC tumors
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S1 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 1 showing NPTX1 is highly expressed in both human and murine highly metastatic PDAC models.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 1 showing NPTX1 is highly expressed in both human and murine highly metastatic PDAC models.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S5 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 5 showing AMIGO2 as a cell surface receptor for NPTX1 and a driver of PDAC metastasis
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 5 showing AMIGO2 as a cell surface receptor for NPTX1 and a driver of PDAC metastasis
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Key Resource Table from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Key resource table with cat numbers
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessKey Resource Table from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Key resource table with cat numbers
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S2 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 2 showing NPTX1 drives liver metastasis of PDAC
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 2 showing NPTX1 drives liver metastasis of PDAC
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S4 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 4 showing NPTX1 promotes cell growth under hypoxic conditions
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S4 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 4 showing NPTX1 promotes cell growth under hypoxic conditions
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S7 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. figure 7 showing therapeutic targeting of NPTX1 with monoclonal NPTX1 antibody
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S7 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. figure 7 showing therapeutic targeting of NPTX1 with monoclonal NPTX1 antibody
- Open Access
- 2 December 2024
- Published by American Association for Cancer Research (AACR)
Figure S5 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 5 showing AMIGO2 as a cell surface receptor for NPTX1 and a driver of PDAC metastasis
- 2 December 2024
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from A Targetable Secreted Neural Protein Drives Pancreatic Cancer Metastatic Colonization and HIF1α Nuclear Retention
Suppl. Figure 5 showing AMIGO2 as a cell surface receptor for NPTX1 and a driver of PDAC metastasis