Lishan Jiang
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Lishan Jiang has a total of 8 co-authors. Here are the top co-authors by number of shared publications:
- Chao Song
Neurologic Disorders and Regenerative Repair Laboratory, Shandong Second Medical University, Weifang 261053, China
1 shared publication - Hang Yu
Neurologic Disorders and Regenerative Repair Laboratory, Shandong Second Medical University, Weifang 261053, China
1 shared publication - Li Yu
School of Basic Medical Sciences, Experimental Center for Medical Research, Neurologic Disorders and Regeneration Repair Lab of Shandong Higher Education, Shandong Second Medical University, Weifang, Shandong 261053, China; Basic Medical College, Binzhou Medical University, Yantai, Shandong 264003, China
1 shared publication - Li Zhou
Neurologic Disorders and Regenerative Repair Laboratory, Shandong Second Medical University, Weifang 261053, China
1 shared publication - Mingxia Shao
Neurologic Disorders and Regenerative Repair Laboratory, Shandong Second Medical University, Weifang 261053, China
1 shared publication - Siqi Wang
Neurologic Disorders and Regenerative Repair Laboratory, Shandong Second Medical University, Weifang 261053, China
1 shared publication
Latest Publications
- Open Access
The Role of Epigenetic Mechanisms in the Development of PM2.5-Induced Cognitive Impairment
PM2.5 is fine particulate matter with a diameter of less than 2.5 μm. Recent evidence has shown that exposure to PM2.5 markedly elevates the risk of neurodegenerative diseases, neurodevelopmental disorders, and cardiovascular diseases, which may culminate in cognitive impairment. Nevertheless, the precise mechanisms through which PM2.5 affects cognitive function are unclear. Recent studies have demonstrated that PM2.5-induced epigenetic alterations are associated with the development of cognitive impairment. Epigenetic alterations include modifications to DNA methylation, histone modifications, and non-coding RNAs. The underlying mechanisms of epigenetic alterations are related to inflammation, synaptic dysfunction, cardiovascular factors, and alterations in neuronal structure and function. This review reports the latest findings on the relationship between PM2.5-induced epigenetic alterations and the development of cognitive disorders, offering novel insights into the cognitive effects of air pollution.
Open AccessThe Role of Epigenetic Mechanisms in the Development of PM2.5-Induced Cognitive Impairment
PM2.5 is fine particulate matter with a diameter of less than 2.5 μm. Recent evidence has shown that exposure to PM2.5 markedly elevates the risk of neurodegenerative diseases, neurodevelopmental disorders, and cardiovascular diseases, which may culminate in cognitive impairment. Nevertheless, the precise mechanisms through which PM2.5 affects cognitive function are unclear. Recent studies have demonstrated that PM2.5-induced epigenetic alterations are associated with the development of cognitive impairment. Epigenetic alterations include modifications to DNA methylation, histone modifications, and non-coding RNAs. The underlying mechanisms of epigenetic alterations are related to inflammation, synaptic dysfunction, cardiovascular factors, and alterations in neuronal structure and function. This review reports the latest findings on the relationship between PM2.5-induced epigenetic alterations and the development of cognitive disorders, offering novel insights into the cognitive effects of air pollution.