Mona Asghariahmadabad
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Mona Asghariahmadabad has a total of 9 co-authors. Here are the top co-authors by number of shared publications:
- Alexander Fadi Haddad
Department of Neurological Surgery, University of California, San Francisco, San Francisco, California, USA
1 shared publication - David Sigmund Liebeskind
Department of Neurology, University of Southern California, Los Angeles, CA
1 shared publication - Jeffrey L. Saver
Department of Neurology, University of California Los Angeles (J.L.S.).
1 shared publication - Kambiz Nael
Department of Radiology & Biomedical Imaging, University of California, San Francisco, California, USA
1 shared publication - Luis Emilio Savastano
Neurological Surgery, University of California San Francisco, San Francisco, California, USA
1 shared publication Marom Bikson
Department of Biomedical Engineering, The City College of New York (CCNY), NY (M.B.).
1 shared publication
Latest Publications
- Open Access
- 1 January 2026
- Published by Wolters Kluwer Health in Stroke: Vascular and Interventional Neurology
- Vol. 6 (1)
Changes in Oxygen Metabolism Biomarkers of Ischemic Tissue Treated With Electrical Stimulation
- 1 January 2026
- Published by Wolters Kluwer Health in Stroke: Vascular and Interventional Neurology
- Vol. 6 (1)
Open AccessChanges in Oxygen Metabolism Biomarkers of Ischemic Tissue Treated With Electrical Stimulation
- Open Access
- 5 April 2024
- Published by Wolters Kluwer Health in Neurosurgery Practice
- Vol. 5 (2) , e00087
Reversal of Middle Cerebral Artery Stenosis by Minimally Invasive Intracerebral Hematoma Evacuation
BACKGROUND AND IMPORTANCE: Acute intracerebral hematomas are known to induce significant mass effects within the brain, leading to critical complications such as cerebral midline shift, herniation, and increased intracranial pressure. The timing and efficacy of intracerebral hematoma evacuation remain subjects of ongoing debate in current literature. CLINICAL PRESENTATION: In our case report, we present a 74-year-old female patient diagnosed with basal ganglia hematoma. The resultant mass effect from the intracerebral hematoma led to middle cerebral artery (MCA) stenosis. Notably, early-stage minimally invasive hematoma evacuation was pivotal in facilitating successful revascularization of the MCA. CONCLUSION: Our case underscores the significance of prompt identification and management of MCA stenosis arising from intracerebral hematoma. Early intervention through minimally invasive hematoma evacuation proved instrumental in achieving successful MCA revascularization. These findings emphasize the critical role of timely interventions in mitigating potential complications associated with intracerebral hematoma.
- 5 April 2024
- Published by Wolters Kluwer Health in Neurosurgery Practice
- Vol. 5 (2) , e00087
Open AccessReversal of Middle Cerebral Artery Stenosis by Minimally Invasive Intracerebral Hematoma Evacuation
BACKGROUND AND IMPORTANCE: Acute intracerebral hematomas are known to induce significant mass effects within the brain, leading to critical complications such as cerebral midline shift, herniation, and increased intracranial pressure. The timing and efficacy of intracerebral hematoma evacuation remain subjects of ongoing debate in current literature. CLINICAL PRESENTATION: In our case report, we present a 74-year-old female patient diagnosed with basal ganglia hematoma. The resultant mass effect from the intracerebral hematoma led to middle cerebral artery (MCA) stenosis. Notably, early-stage minimally invasive hematoma evacuation was pivotal in facilitating successful revascularization of the MCA. CONCLUSION: Our case underscores the significance of prompt identification and management of MCA stenosis arising from intracerebral hematoma. Early intervention through minimally invasive hematoma evacuation proved instrumental in achieving successful MCA revascularization. These findings emphasize the critical role of timely interventions in mitigating potential complications associated with intracerebral hematoma.