Katja E. Wartenberg
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Katja E. Wartenberg has a total of 5 co-authors. Here are the top co-authors by number of shared publications:
Andreas Funk
Department of General Surgery, Klinikum Altenburger Land, Am Waldessaum 10, 04600 Altenburg, Germany
1 shared publicationAndreas Meyer
Central Laboratory, Klinikum Altenburger Land, Am Waldessaum 10, 04600 Altenburg, Germany
1 shared publicationAnett Stoll
Department of Neurology, Klinikum Altenburger Land, Am Waldessaum 10, 04600 Altenburg, Germany
1 shared publicationJ. Michael Schmidt
Division of Neurocritical Care, Neurological Institute, Columbia University College of Physicians and Surgeons, 710 West 168th Street, New York, NY 10032, USA
1 shared publicationJoerg Berrouschot
Department of Neurology, Klinikum Altenburger Land, Am Waldessaum 10, 04600 Altenburg, Germany
1 shared publication
Latest Publications
- Open Access
- 1 January 2011
- Published by Wiley in Stroke Research and Treatment
- Vol. 2011, 1-8
Infection after Acute Ischemic Stroke: Risk Factors, Biomarkers, and Outcome
Background. The activation of inflammatory cascades triggered by ischemic stroke may play a key role in the development of infections.Methods. Patients admitted with ischemic stroke within 24 hours were prospectively enrolled. Biomarkers of infection were measured on days 1, 3, and 5. The patients were continuously monitored for predefined infections.Results. Patients with infection were older (OR 1.06 per year, 95% CI 1.01–1.11) and had a higher National Institute of Health Stroke Scale Score (NIHSS, OR 1.21, 95% CI 1.10–1.34), localization in the insula, and higher stroke volumes on diffusion-weighted imaging. The maximum temperature on days 1 and 3, leukocytes, interleukin-6, lipopolysaccharide-binding protein on days 1, 3, and 5, C-reactive protein on days 3 and 5, and procalcitonin on day 5 were higher and HLA-DR-expression on monocytes on days 1, 3, and 5 lower in patients with infection. Age and NIHSS predicted the development of infections. Infection was an independent predictor of poor functional outcome.Conclusions. Severe stroke and increasing age were shown to be early predictors for infections after stroke.
- 1 January 2011
- Published by Wiley in Stroke Research and Treatment
- Vol. 2011, 1-8
Open AccessInfection after Acute Ischemic Stroke: Risk Factors, Biomarkers, and Outcome
Background. The activation of inflammatory cascades triggered by ischemic stroke may play a key role in the development of infections.Methods. Patients admitted with ischemic stroke within 24 hours were prospectively enrolled. Biomarkers of infection were measured on days 1, 3, and 5. The patients were continuously monitored for predefined infections.Results. Patients with infection were older (OR 1.06 per year, 95% CI 1.01–1.11) and had a higher National Institute of Health Stroke Scale Score (NIHSS, OR 1.21, 95% CI 1.10–1.34), localization in the insula, and higher stroke volumes on diffusion-weighted imaging. The maximum temperature on days 1 and 3, leukocytes, interleukin-6, lipopolysaccharide-binding protein on days 1, 3, and 5, C-reactive protein on days 3 and 5, and procalcitonin on day 5 were higher and HLA-DR-expression on monocytes on days 1, 3, and 5 lower in patients with infection. Age and NIHSS predicted the development of infections. Infection was an independent predictor of poor functional outcome.Conclusions. Severe stroke and increasing age were shown to be early predictors for infections after stroke.