Xiyi Lu
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Xiyi Lu has a total of 8 co-authors. Here are the top co-authors by number of shared publications:
- Chen Zhang
Department of Oncology, First Affiliated Hospital of Nanjing Medical University, Nanjing, China
21 shared publications - Dandan Yin
Clinical Research Center, The Second Hospital of Nanjing, Nanjing University of Chinese Medicine, Nanjing, China
21 shared publications - Erbao Zhang
Department of Epidemiology, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, China
21 shared publications - Jiali Dai
Department of Oncology, First Affiliated Hospital of Nanjing Medical University, Nanjing, China
21 shared publications - Jiali Xu
Department of Oncology, First Affiliated Hospital of Nanjing Medical University, Nanjing, China
21 shared publications - Lei Song
Department of Oncology, First Affiliated Hospital of Nanjing Medical University, Nanjing, China
21 shared publications
Latest Publications
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S2 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
The effect of MYLK-AS1 expression on gefitinib resistance of NSCLC.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
The effect of MYLK-AS1 expression on gefitinib resistance of NSCLC.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S10 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
MYLK-AS1 promotes TKI resistance of lung cancer by regulating GLUD1.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S10 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
MYLK-AS1 promotes TKI resistance of lung cancer by regulating GLUD1.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Movie S1 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
The quick rearrangement of ILF3 droplets. A time-lapse movie of droplets formed by ILF3 undergoes fusion event.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessMovie S1 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
The quick rearrangement of ILF3 droplets. A time-lapse movie of droplets formed by ILF3 undergoes fusion event.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Table S5 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
A list of top five potential MYLK-AS1-interacting protein candidates in PC9/GR cells based on RNA-protein pull-down assays and mass spectrometry analysis.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S5 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
A list of top five potential MYLK-AS1-interacting protein candidates in PC9/GR cells based on RNA-protein pull-down assays and mass spectrometry analysis.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S5 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
METTL3 induces the expression of MYLK-AS1 and promotes TKI resistance.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
METTL3 induces the expression of MYLK-AS1 and promotes TKI resistance.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Table S3 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
Oligonucleotides sequences summary.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S3 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
Oligonucleotides sequences summary.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S9 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
MYLK-AS1 enhances the stability of GLUD1 mRNA by directly binding to ILF3.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S9 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
MYLK-AS1 enhances the stability of GLUD1 mRNA by directly binding to ILF3.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S8 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
MYLK-AS1 drives phase separation of ILF3 in PC9/OR.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S8 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
MYLK-AS1 drives phase separation of ILF3 in PC9/OR.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S6 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
METTL3-mediated post-transcriptional m6A modification contributes to the activation of MYLK-AS1 in TKI resistance of NSCLC.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S6 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
METTL3-mediated post-transcriptional m6A modification contributes to the activation of MYLK-AS1 in TKI resistance of NSCLC.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Table S4 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
MRNAs variation abundance (>=1.5-fold) in MYLK-AS1-knockdown PC9/GR cells.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S4 from MYLK-AS1 Enhances Glutamine Metabolism to Promote EGFR Inhibitor Resistance in Non–Small Cell Lung Cancer
MRNAs variation abundance (>=1.5-fold) in MYLK-AS1-knockdown PC9/GR cells.