Giulia Montuori
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Giulia Montuori has a total of 35 co-authors. Here are the top co-authors by number of shared publications:
- Angelika Eggert
Department of Pediatric Oncology/Hematology, Charité-Universitätsmedizin Berlin, Berlin, Germany
13 shared publications - Annette Künkele
Department of Pediatric Oncology and Hematology, Charité – Universitätsmedizin Berlin, Berlin, Germany
13 shared publications - Annika Lehmann
Institute of Pathology, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt Universität zu Berlin, Berlin, Germany
13 shared publications - Anton George Henssen
Department of Pediatric Hematology and Oncology, Charité-Universitätsmedizin Berlin, Berlin, Germany
13 shared publications - Arend Koch
Department of Neuropathology Charité‐Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin and Humboldt Universität zu Berlin Berlin Germany
13 shared publications - Bartolomeo Bosco
Department of Pediatric Oncology/Hematology, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt Universität zu Berlin, Berlin, Germany
13 shared publications
Latest Publications
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Figure S6 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S6 shows copy number dynamics in EGFR-amplified glioblastoma samples
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S6 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S6 shows copy number dynamics in EGFR-amplified glioblastoma samples
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Figure S1 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S1 characterises the MYCN amplification status, copy number heterogeneity and growth behaviour in neuroblastoma samples
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S1 characterises the MYCN amplification status, copy number heterogeneity and growth behaviour in neuroblastoma samples
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Table S4 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Table S4 contains information about the MYCN status of neuroblastoma patient samples used in Figure 1 and Supplementary Figure 1
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S4 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Table S4 contains information about the MYCN status of neuroblastoma patient samples used in Figure 1 and Supplementary Figure 1
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Figure S2 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S2 shows simulation results of clonogenic assays under copy number-dependent and -independent cell fitness
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S2 shows simulation results of clonogenic assays under copy number-dependent and -independent cell fitness
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Figure S3 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S3 demonstrates how MYC(N) dosage differences drive phenotypic heterogeneity in ecDNA-containing cancers
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S3 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S3 demonstrates how MYC(N) dosage differences drive phenotypic heterogeneity in ecDNA-containing cancers
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Table S1 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Table S1 contains FISH proteomics datasets
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S1 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Table S1 contains FISH proteomics datasets
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Figure S4 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S4 provides computational and experimental models of ecDNA-dependent treatment responses in neuroblastoma
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S4 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S4 provides computational and experimental models of ecDNA-dependent treatment responses in neuroblastoma
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Table S2 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Table S2 lists the GDCS compounds used in Supplementary Figure S4L
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S2 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Table S2 lists the GDCS compounds used in Supplementary Figure S4L
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Figure S8 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S8 illustrates how one-two punch, senolytic therapies can be used to target tumor cells with low MYCN copy numbers
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S8 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S8 illustrates how one-two punch, senolytic therapies can be used to target tumor cells with low MYCN copy numbers
- Open Access
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Figure S7 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S7 demonstrates the impact of MYCN dosage on treatment response in neuroblastoma
- 6 October 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S7 from Extrachromosomal DNA–Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers
Supplementary Figure S7 demonstrates the impact of MYCN dosage on treatment response in neuroblastoma