Paloma Guernalec
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Paloma Guernalec has a total of 30 co-authors. Here are the top co-authors by number of shared publications:
- Barbara D.S. Passaia
Department of Computational Biology, St. Jude Children’s Research Hospital, Memphis, Tennessee
20 shared publications - Bensheng Ju
Department of Computational Biology, St. Jude Children’s Research Hospital, Memphis, Tennessee
20 shared publications - Brian J. Abraham
Department of Computational Biology, St. Jude Children’s Research Hospital, Memphis, USA
20 shared publications - Chunliang Li
Department of Tumor Cell Biology, St. Jude Children’s Research Hospital, Memphis, USA
20 shared publications - Declan Bennett
Department of Computational Biology, St. Jude Children’s Research Hospital, Memphis, TN
20 shared publications - Flore Cuisin
Department of Tumor Cell Biology, St. Jude Children’s Research Hospital, Memphis, Tennessee
20 shared publications
Latest Publications
- Open Access
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S2 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
AmpliconArchitect reconstructions of MYC-amplified G3-MB cell models.
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S2 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
AmpliconArchitect reconstructions of MYC-amplified G3-MB cell models.
- Open Access
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S4 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
Post-MAGeCK analysis CRISPRi screen results.
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S4 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
Post-MAGeCK analysis CRISPRi screen results.
- Open Access
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S8 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
AmpliconArchitect reconstructions of D425 ecDNA after ecMYC E1 silencing.
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S8 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
AmpliconArchitect reconstructions of D425 ecDNA after ecMYC E1 silencing.
- Open Access
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S2 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
List of sgRNA sequences for validation experiments.
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S2 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
List of sgRNA sequences for validation experiments.
- Open Access
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S1 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
Dependency Map results for MYC and OTX2 in the G3-MB D425 and D458 cell lines.
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S1 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
Dependency Map results for MYC and OTX2 in the G3-MB D425 and D458 cell lines.
- Open Access
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S6 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
PVT1 promoter and MYC transcription in ecDNA-amplified G3-MB.
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S6 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
PVT1 promoter and MYC transcription in ecDNA-amplified G3-MB.
- Open Access
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
List of primary and secondary antibodies.
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
List of primary and secondary antibodies.
- Open Access
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S3 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
List of sgRNA sequences for the CRISPR inhibition screen of D425.
- 2 July 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S3 from A Conserved Enhancer Locus in Extrachromosomal DNA and Homogeneously Staining Regions Activates MYC Transcription in Group 3 Medulloblastoma
List of sgRNA sequences for the CRISPR inhibition screen of D425.