Juliannie Herrera
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Juliannie Herrera has a total of 41 co-authors. Here are the top co-authors by number of shared publications:
- Aniko Szabo
Medical College of Wisconsin
21 shared publications - Daniel Dorbin
Medical College of Wisconsin 1 Department of Biochemistry, LaBahn Pancreatic Cancer Program Milwaukee, Wisconsin
21 shared publications - Douglas B. Evans
Department of Surgery, Medical College of Wisconsin, Milwaukee, WI 53226, USA
21 shared publications - G. Aaron Hobbes
Department of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina
21 shared publications - John Fairbanks Langenheim
Thomas Jefferson University 6 Department of Pharmacology, Physiology, and Cancer Biology Philadelphia, Pennsylvania
21 shared publications - Mandana Kamgar
Mary Ann and Charles LaBahn Pancreatic Cancer Program, Medical College of Wisconsin, Milwaukee, WI 53226, USA
21 shared publications
Latest Publications
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Figure S1 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S1. Role of KRASMUT-SOS axis in MAPK signaling
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S1. Role of KRASMUT-SOS axis in MAPK signaling
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Figure S2 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S2. Ex-vivo organotypic slice culture methodology
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S2. Ex-vivo organotypic slice culture methodology
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Figure S3 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S3. RAS inhibitor resistance
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S3 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S3. RAS inhibitor resistance
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Figure S5 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S5. Characterization of toroid integrity by orthogonal methods
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S5. Characterization of toroid integrity by orthogonal methods
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Figure S4 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S4 Patient Tumor Bioprinting
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S4 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S4 Patient Tumor Bioprinting
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Figure S6 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S6. Sensitivity to trametinib
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S6 from Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer
Figure S6. Sensitivity to trametinib
- Open Access
- 31 August 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 2 from Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma
Mutant KRAS allele dependent activation of WT-RAS
- 31 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 2 from Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma
Mutant KRAS allele dependent activation of WT-RAS
- Open Access
- 31 August 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 3 from Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma
Kaplan-Meier estimates of progression-free survival
- 31 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 3 from Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma
Kaplan-Meier estimates of progression-free survival
- Open Access
- 31 August 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 1 from Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma
Supporting Data for Isogenic cells
- 31 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 1 from Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma
Supporting Data for Isogenic cells
- Open Access
- 31 August 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table 1 from Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma
Patient, tumor and treatment characteristics
- 31 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table 1 from Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma
Patient, tumor and treatment characteristics