Yuan Lu
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Yuan Lu has a total of 7 co-authors. Here are the top co-authors by number of shared publications:
- Cunwu Zuo
College of Horticulture, Gansu Agricultural University, Lanzhou 730070, China
1 shared publication - E. Sun
College of Horticulture, Gansu Agricultural University, Lanzhou 730070, China
1 shared publication - Hongqiang Yu
College of Horticulture, Gansu Agricultural University, Lanzhou 730070, China
1 shared publication - Junying Zhao
Gansu Academy of Forestry Sciences, Lanzhou 730020, China
1 shared publication - Longgang Zuo
Gansu Academy of Forestry Sciences, Lanzhou 730020, China
1 shared publication - Minrui Cai
College of Horticulture, Gansu Agricultural University, Lanzhou 730070, China
1 shared publication
Latest Publications
- Open Access
- 23 December 2025
- Published by MDPI AG in Current Issues in Molecular Biology
- Vol. 48 (1) , 14
Jasmonic Acid Signals Involved in Valsa Canker Resistance Caused by C2H2-Type Transcription Factor PbeSTOP2 in Pyrus betulifolia
Valsa canker, a destructive necrotrophic disease caused by Valsa species (V. mali and V. pyri), threatens global apple and pear production. Resistance breeding has gained much attention because of its environmental friendliness and effectiveness, making it extremely critical to obtain key disease resistance genes. In this study, we identified that the expression of PbeSTOP2, a C2H2-type transcription factor in ‘Duli-G03’ (Pyrus betulifolia, rootstock of pear), was induced in response to signals from Vp and Vp metabolites (VpM). Transient expression in fruits and stable overexpression in suspension cells demonstrated that PbeSTOP2 enhances resistance to Vp. In overexpressing cells, genes associated with jasmonic acid (JA) and pattern-triggered immunity (PTI) signaling were upregulated, and endogenous JA and auxin (IAA) levels increased. These results revealed that JA signaling was involved in the enhanced Valsa canker resistance conferred by PbeSTOP2 overexpression. This finding on host disease resistance should facilitate the breeding efforts for Valsa canker resistance.
- 23 December 2025
- Published by MDPI AG in Current Issues in Molecular Biology
- Vol. 48 (1) , 14
Open AccessJasmonic Acid Signals Involved in Valsa Canker Resistance Caused by C2H2-Type Transcription Factor PbeSTOP2 in Pyrus betulifolia
Valsa canker, a destructive necrotrophic disease caused by Valsa species (V. mali and V. pyri), threatens global apple and pear production. Resistance breeding has gained much attention because of its environmental friendliness and effectiveness, making it extremely critical to obtain key disease resistance genes. In this study, we identified that the expression of PbeSTOP2, a C2H2-type transcription factor in ‘Duli-G03’ (Pyrus betulifolia, rootstock of pear), was induced in response to signals from Vp and Vp metabolites (VpM). Transient expression in fruits and stable overexpression in suspension cells demonstrated that PbeSTOP2 enhances resistance to Vp. In overexpressing cells, genes associated with jasmonic acid (JA) and pattern-triggered immunity (PTI) signaling were upregulated, and endogenous JA and auxin (IAA) levels increased. These results revealed that JA signaling was involved in the enhanced Valsa canker resistance conferred by PbeSTOP2 overexpression. This finding on host disease resistance should facilitate the breeding efforts for Valsa canker resistance.