Salma Abid
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Salma Abid has a total of 9 co-authors. Here are the top co-authors by number of shared publications:
- Amal Miraoui
Pediatric Intensive Care Unit, University of Tunis of EL-Manar, Faculty of Medicine of Tunis, Children’s Hospital Bechir Hamza of Tunis, Tunis, Tunisia
1 shared publication - Ameni Sallemi
Laboratory of Clinical Pharmacology, National Center Chalbibelkahia of Pharmacovigilance of Tunis, 9 Avenue Dr. Zouhaier Essafi 1006 Bab Souika, Tunis 1007, Tunisia
1 shared publication - Ichrak Landolsi
Charles Nicolle Hospital, Laboratory of Microbiology, National Influenza Centre, National Reference Laboratory for Influenza and Other Respiratory Viruses, Tunis 1006, Tunisia
1 shared publication - Ilhem Boutiba-Ben Boubaker
Department of Microbiology, Charles Nicolle Hospital, Tunis 1006, Tunisia
1 shared publication - Khaled Menif
Pediatric Intensive Care Unit, Béchir Hamza Children’s Hospital, Tunis 1006, Tunisia
1 shared publication - Lamia Kanzari
LR99ES09, Faculty of Medicine of Tunis, University of Tunis El Manar, Tunis 1007, Tunisia
1 shared publication
Latest Publications
- Open Access
- 30 April 2026
- Published by MDPI AG in Microorganisms
- Vol. 14 (5) , 1009
Intra-Omicron Reinfection with JN.1.16 and NB.1.8.1 in a Preterm Infant: First NB.1.8.1 Detection in Tunisia—A Case Report
Highly mutated Omicron sub-lineages JN.1 and NB.1.8.1 harbor extensive spike changes, but their impact in preterm infants is poorly documented. We report a preterm male infant with three hospitalizations in seven weeks: severe SARS-CoV-2 ARDS at 40 days of life (DOL 40) requiring ventilation caused by JN.1.16, HCoV-OC43 infection at DOL 65, and a mild SARS-CoV-2 reinfection at DOL 87 due to NB.1.8.1, the first detection of this variant in Tunisia. Spike analysis showed a shared JN.1 backbone but distinct N-terminal and receptor-binding domain changes, supporting intra-Omicron reinfection driven by antigenic divergence and immature immunity and underscoring the value of pediatric genomic surveillance, including phylogenetic placement of case genomes within local Omicron diversity.
- 30 April 2026
- Published by MDPI AG in Microorganisms
- Vol. 14 (5) , 1009
Open AccessIntra-Omicron Reinfection with JN.1.16 and NB.1.8.1 in a Preterm Infant: First NB.1.8.1 Detection in Tunisia—A Case Report
Highly mutated Omicron sub-lineages JN.1 and NB.1.8.1 harbor extensive spike changes, but their impact in preterm infants is poorly documented. We report a preterm male infant with three hospitalizations in seven weeks: severe SARS-CoV-2 ARDS at 40 days of life (DOL 40) requiring ventilation caused by JN.1.16, HCoV-OC43 infection at DOL 65, and a mild SARS-CoV-2 reinfection at DOL 87 due to NB.1.8.1, the first detection of this variant in Tunisia. Spike analysis showed a shared JN.1 backbone but distinct N-terminal and receptor-binding domain changes, supporting intra-Omicron reinfection driven by antigenic divergence and immature immunity and underscoring the value of pediatric genomic surveillance, including phylogenetic placement of case genomes within local Omicron diversity.