Four Human Ras Homologs Differ in Their Abilities to Activate Raf-1, Induce Transformation, and Stimulate Cell Motility
Open Access
- 1 June 1999
- journal article
- research article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 274 (24) , 17164-17170
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Increasing complexity of Ras signalingOncogene, 1998
- CAS/Crk Coupling Serves as a “Molecular Switch” for Induction of Cell MigrationJournal of Cell Biology, 1998
- Developmentally-regulated expression of murine K-ras isoformsOncogene, 1997
- Phosphorylation-dependent activation of the Ras-GRF/CDC25Mm exchange factor by muscarinic receptors and G-protein βγ subunitsNature, 1996
- Cell Migration: A Physically Integrated Molecular ProcessCell, 1996
- Calcium activation of Ras mediated by neuronal exchange factor Ras-GRFNature, 1995
- Specificity of receptor tyrosine kinase signaling: Transient versus sustained extracellular signal-regulated kinase activationCell, 1995
- ras GENESAnnual Review of Biochemistry, 1987
- Structure of the Ki-ras gene of the human lung carcinoma cell line Calu-1Nature, 1983
- Activation of Ki-ras2 gene in human colon and lung carcinomas by two different point mutationsNature, 1983