Role of HNF4α-cMyc Interaction in CDE Diet–Induced Liver Injury and Regeneration
Open Access
- 6 April 2024
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 194 (7) , 1218-1229
Abstract
No abstract availableKeywords
All Related Versions
Funding Information
- NIDDK
- National Institutes of Health (R01 DK0198414, R56 DK112768)
This publication has 18 references indexed in Scilit:
- Progressive loss of hepatocyte nuclear factor 4 alpha activity in chronic liver diseases in humansHepatology, 2022
- Hepatocyte Nuclear Factor 4 Alpha Activation Is Essential for Termination of Liver Regeneration in MiceHepatology, 2018
- Targeting oncogenic Myc as a strategy for cancer treatmentSignal Transduction and Targeted Therapy, 2018
- Enhanced expression of c-myc in hepatocytes promotes initiation and progression of alcoholic liver diseaseJournal of Hepatology, 2016
- Overexpression of c-myc in hepatocytes promotes activation of hepatic stellate cells and facilitates the onset of liver fibrosisBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2013
- Hepatocyte-specific deletion of farnesoid X receptor delays but does not inhibit liver regeneration after partial hepatectomy in miceHepatology, 2012
- Liver progenitor cell markers correlate with liver damage and predict short-term mortality in patients with alcoholic hepatitisHepatology, 2012
- Ductular reactions in human liver: Diversity at the interfaceHepatology, 2011
- Relation between liver progenitor cell expansion and extracellular matrix deposition in a CDE-induced murine model of chronic liver injury #Hepatology, 2009
- Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT MethodMethods, 2001