A BTLA-Mediated Bait and Switch Strategy Permits Listeria Expansion in CD8α+ DCs to Promote Long-Term T Cell Responses
- 1 July 2014
- journal article
- Published by Elsevier in Cell Host & Microbe
- Vol. 16 (1) , 68-80
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- B Cell Maintenance of Subcapsular Sinus Macrophages Protects against a Fatal Viral Infection Independent of Adaptive ImmunityImmunity, 2012
- Critical role for perforin and Fas-dependent killing of dendritic cells in the control of inflammationBlood, 2012
- Distinct responses of splenic dendritic cell subsets to infection with Listeria monocytogenes: Maturation phenotype, level of infection, and T cell priming capacity ex vivoCellular Immunology, 2011
- Inflammatory Monocytes and Neutrophils Are Licensed to Kill during Memory Responses In VivoPLOS Pathogens, 2011
- A platelet-mediated system for shuttling blood-borne bacteria to CD8α+ dendritic cells depends on glycoprotein GPIb and complement C3Nature Immunology, 2011
- CD8α+ Dendritic Cells Are an Obligate Cellular Entry Point for Productive Infection by Listeria monocytogenesImmunity, 2011
- B and T Lymphocyte Attenuator Tempers Early Infection ImmunityThe Journal of Immunology, 2009
- Batf3 Deficiency Reveals a Critical Role for CD8α + Dendritic Cells in Cytotoxic T Cell ImmunityScience, 2008
- Elimination of Antigen-Presenting Cells and Autoreactive T Cells by Fas Contributes to Prevention of AutoimmunityImmunity, 2007
- BTLA is a lymphocyte inhibitory receptor with similarities to CTLA-4 and PD-1Nature Immunology, 2003