Abstract
VO2NPs trigger endoplasmic reticulum stress, release of calcium ions, and overproduction of ROS, leading to a significant proliferation inhibition and apoptosis in macrophages. VO2NPs display higher responses and toxicity than bulk VO2.
Funding Information
  • National Natural Science Foundation of China (22071145, 31871007, 32071404)
  • National Key Research and Development Program of China (2016YFA0201600)

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