Profilin-2 increased expression and its altered interaction with β-actin in the striatum of 3-nitropropionic acid-induced Huntington’s disease in rats
- 1 December 2014
- journal article
- Published by Elsevier in Neuroscience
- Vol. 281, 216-228
Abstract
No abstract availableKeywords
Funding Information
- Department of Science and Technology
- Govt. of India vide (SR/FTP/LS-A-61/2001)
- Neurodegenerative Diseases: Causes and Corrections (BSC-0115)
This publication has 46 references indexed in Scilit:
- Fine-tuning of neuronal architecture requires two profilin isoformsProceedings of the National Academy of Sciences, 2010
- Striatal dopamine level contributes to hydroxyl radical generation and subsequent neurodegeneration in the striatum in 3-nitropropionic acid-induced Huntington's disease in ratsNeurochemistry International, 2009
- Rhes, a Striatal Specific Protein, Mediates Mutant-Huntingtin CytotoxicityScience, 2009
- Complex II inhibition by 3-NP causes mitochondrial fragmentation and neuronal cell death via an NMDA- and ROS-dependent pathwayCell Death & Differentiation, 2009
- Phosphorylation of Profilin by ROCK1 Regulates Polyglutamine AggregationMolecular and Cellular Biology, 2008
- Expression of expanded polyglutamine targets profilin for degradation and alters actin dynamicsNeurobiology of Disease, 2008
- Dopaminergic Signaling and Striatal Neurodegeneration in Huntington's DiseaseThe Journal of Neuroscience, 2007
- Profilin2 contributes to synaptic vesicle exocytosis, neuronal excitability, and novelty‐seeking behaviorThe EMBO Journal, 2007
- Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT MethodMethods, 2001
- Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in BrainScience, 1997