Blockade of receptor for advanced glycation end products protects against systolic overload-induced heart failure after transverse aortic constriction in mice

This publication has 40 references indexed in Scilit:

• Prognosis of patients with heart failure and reduced ejection fraction in China
  Experimental and Therapeutic Medicine, 2013
• The Hypoxia-Inducible MicroRNA Cluster miR-199a∼214 Targets Myocardial PPARδ and Impairs Mitochondrial Fatty Acid Oxidation
  Cell Metabolism, 2013
• Functional Role of Soluble Receptor for Advanced Glycation End Products in Stroke
  Arteriosclerosis, Thrombosis, and Vascular Biology, 2013
• Cardiomyocyte-specific IκB kinase (IKK)/NF-κB activation induces reversible inflammatory cardiomyopathy and heart failure
  Proceedings of the National Academy of Sciences, 2012
• RAGE signaling mediates post-injury arterial neointima formation by suppression of liver kinase B1 and AMPK activity
  Atherosclerosis, 2012
• Cardiomyocyte NF-κB p65 promotes adverse remodelling, apoptosis, and endoplasmic reticulum stress in heart failure
  Cardiovascular Research, 2010
• Energetics and metabolism in the failing heart: important but poorly understood
  Current Opinion in Clinical Nutrition and Metabolic Care, 2010
• RAGE Modulates Hypoxia/Reoxygenation Injury in Adult Murine Cardiomyocytes via JNK and GSK-3β Signaling Pathways
  PLOS ONE, 2010
• Chronic NF-κB blockade reduces cytosolic and mitochondrial oxidative stress and attenuates renal injury and hypertension in SHR
  American Journal of Physiology-Renal Physiology, 2009
• AMP-activated protein kinase (AMPK) action in skeletal muscle via direct phosphorylation of PGC-1α
  Proceedings of the National Academy of Sciences, 2007