Overexpression of Metallothionein-II Sensitizes Rodent Cells to Apoptosis Induced by DNA Cross-linking Agent through Inhibition of NF-κB Activation
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Open Access
- 1 February 2002
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 277 (7) , 4764-4769
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Involvement of apoptosis in mitomycin C hypersensitivity of Chinese hamster cell mutantsBiochemical Pharmacology, 2000
- Lack of correlation between repair of DNA interstrand cross-links and hypersensitivity of hamster cells towards mitomycin C and cisplatin.FEBS Letters, 1998
- The elusive function of metallothioneinsProceedings of the National Academy of Sciences, 1998
- The mitomycin bioreductive antitumor agents: Cross-linking and alkylation of DNA as the molecular basis of their activityPharmacology & Therapeutics, 1997
- The p53 Status of Chinese Hamster V79 Cells Frequently Used for Studies on DNA Damage and DNA RepairNucleic Acids Research, 1997
- Suppression subtractive hybridization: a method for generating differentially regulated or tissue-specific cDNA probes and libraries.Proceedings of the National Academy of Sciences, 1996
- Genetic analysis of mitomycin C-hypersensitive Chinese hamster cell mutantsMutagenesis, 1994
- Role of the human ERCC-1 gene in gene-specific repair of cisplatin-induced DNA damageNucleic Acids Research, 1994
- The Chinese hamster V79 cell mutant V-H4 is phenotypically like Fanconi anemia cellsSomatic Cell and Molecular Genetics, 1990
- Mutagen-sensitive cell lines are obtained with high frequency in V79 Chinese cellsMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1987