Bing Liu
Research Subjects
Research Interests
Bing Liu has a total of 11 co-authors. Here are the top co-authors by number of shared publications:
- Bai Cui
Institute of Cancer Stem Cell, Dalian Medical University, 9 West Section, Lvshun South Road, Dalian, Liaoning Province, 116044, China
1 shared publication - Fei-Meng Zheng
Department of Medical Oncology of the Eastern Hospital, the First Affiliated Hospital, Sun Yat-Sen University, Guangzhou, China
1 shared publication - Hefei Wang1 shared publication
- Jialiang Zhang1 shared publication
- Jie-Ren Peng
Department of Otorhinolaryngology, Second Affiliated Hospital, Sun Yat-sen University, Guangzhou 510120, China
1 shared publication - Jing Wang
Sun Yat-sen University Cancer Center 7 Department of Basic Research, State Key Laboratory of Oncology in South China, Guangdong Provincial Clinical Research Center for Cancer Guangzhou, China
1 shared publication
Latest Publications
- Open Access
- 26 November 2014
- Published by Springer Nature in Molecular Cancer
- Vol. 13 (1) , 257
SOX1 down-regulates β-catenin and reverses malignant phenotype in nasopharyngeal carcinoma
Aberrant activation of the Wnt/beta-catenin signaling pathway is an important factor in the development of nasopharyngeal carcinoma (NPC). Previous studies have demonstrated that the developmental gene sex-determining region Y (SRY)-box 1 (SOX1) inhibits cervical and liver tumorigenesis by interfering with the Wnt/-catenin signaling pathway. However, the role of SOX1 in NPC remains unclear. This study investigates the function of SOX1 in NPC pathogenesis.
- 26 November 2014
- Published by Springer Nature in Molecular Cancer
- Vol. 13 (1) , 257
Open AccessSOX1 down-regulates β-catenin and reverses malignant phenotype in nasopharyngeal carcinoma
Aberrant activation of the Wnt/beta-catenin signaling pathway is an important factor in the development of nasopharyngeal carcinoma (NPC). Previous studies have demonstrated that the developmental gene sex-determining region Y (SRY)-box 1 (SOX1) inhibits cervical and liver tumorigenesis by interfering with the Wnt/-catenin signaling pathway. However, the role of SOX1 in NPC remains unclear. This study investigates the function of SOX1 in NPC pathogenesis.