Marita Schrenk
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Marita Schrenk has a total of 11 co-authors. Here are the top co-authors by number of shared publications:
- Achim Stephan
Functional Genome Analysis, Deutsches Krebsforschungszentrum (DKFZ), Im Neuenheimer Feld 580, 69120, Heidelberg, Germany
1 shared publication - Aleena Gladkikh
Faculty of Biology, Lomonosov Moscow State University, Leninskie Gory, 1, Bld. 12, 119234 Moscow, Russia
1 shared publication - Evgeny A. Moskalev
Institute of Pathology, University Hospital Erlangen, Friedrich-Alexander University Erlangen-Nuremberg, Erlangen, Germany
1 shared publication - Ivan A. Vorobjev
Department of Biology, School of Sciences and Humanities, Nazarbayev University, Astana, 010000, Kazakhstan
1 shared publication - Jörg D. Hoheisel
Functional Genome Analysis, German Cancer Research Center (DKFZ), Im Neuenheimer Feld 580, 69120 Heidelberg, Germany
1 shared publication - Kamil’ Daniyalovich Kaplanov
Botkin Hospital, Moscow Healthcare Department
1 shared publication
Latest Publications
- Open Access
- 6 June 2012
- Published by Springer Nature in BMC Cancer
- Vol. 12 (1) , 213
Concurrent epigenetic silencing of wnt/β-catenin pathway inhibitor genes in B cell chronic lymphocytic leukaemia
The Wnt/β-catenin signalling is aberrantly activated in primary B cell chronic lymphocytic leukaemia (CLL). Epigenetic silencing of pathway inhibitor genes may be a mechanism for its activation. In this study, we investigated systematically and quantitatively the methylation status of 12 Wnt/β-catenin pathway inhibitor genes – CDH1, DACT1, DKK1, DKK2, DKK3, DKK4, SFRP1, SFRP2, SFRP3, SFRP4, SFRP5 and WIF1 – in the cell lines EHEB and MEC-1 as well as patient samples.
- 6 June 2012
- Published by Springer Nature in BMC Cancer
- Vol. 12 (1) , 213
Open AccessConcurrent epigenetic silencing of wnt/β-catenin pathway inhibitor genes in B cell chronic lymphocytic leukaemia
The Wnt/β-catenin signalling is aberrantly activated in primary B cell chronic lymphocytic leukaemia (CLL). Epigenetic silencing of pathway inhibitor genes may be a mechanism for its activation. In this study, we investigated systematically and quantitatively the methylation status of 12 Wnt/β-catenin pathway inhibitor genes – CDH1, DACT1, DKK1, DKK2, DKK3, DKK4, SFRP1, SFRP2, SFRP3, SFRP4, SFRP5 and WIF1 – in the cell lines EHEB and MEC-1 as well as patient samples.