Victor I. Band
Research Subjects
Research Interests
Victor I. Band has a total of 8 co-authors. Here are the top co-authors by number of shared publications:
- Aroon T. Chande
Georgia Institute of Technology
1 shared publication - David S. Weiss
Emory University School of Medicine
1 shared publication - Eileen M. Burd
Department of Pathology and Laboratory Medicine, Emory University School of Medicine
1 shared publication - I. King Jordan
School of Biological Sciences, Georgia Institute of Technology, Atlanta, USA
1 shared publication - Jesse T. Jacob
Emory University School of Medicine
1 shared publication - Jessie E. Wozniak
Emory Vaccine Center, Atlanta, GA 30317, USA
1 shared publication
Latest Publications
- Open Access
- 19 November 2020
- Published by MDPI AG in Antibiotics
- Vol. 9 (11) , 825
Absence of mgrB Alleviates Negative Growth Effects of Colistin Resistance in Enterobacter cloacae
Colistin is an important last-line antibiotic to treat highly resistant Enterobacter infections. Resistance to colistin has emerged among clinical isolates but has been associated with a significant growth defect. Here, we describe a clinical Enterobacter isolate with a deletion of mgrB, a regulator of colistin resistance, leading to high-level resistance in the absence of a growth defect. The identification of a path to resistance unrestrained by growth defects suggests colistin resistance could become more common in Enterobacter.
- 19 November 2020
- Published by MDPI AG in Antibiotics
- Vol. 9 (11) , 825
Open AccessAbsence of mgrB Alleviates Negative Growth Effects of Colistin Resistance in Enterobacter cloacae
Colistin is an important last-line antibiotic to treat highly resistant Enterobacter infections. Resistance to colistin has emerged among clinical isolates but has been associated with a significant growth defect. Here, we describe a clinical Enterobacter isolate with a deletion of mgrB, a regulator of colistin resistance, leading to high-level resistance in the absence of a growth defect. The identification of a path to resistance unrestrained by growth defects suggests colistin resistance could become more common in Enterobacter.