Wenge Zhang

Urban and suburban PM_2.5 samples were collected simultaneously during selected periods representing each season in 2019 in Zibo, China. Samples were analysed for water-soluble inorganic ions, carbon components, and elements. A chemical mass balance model and health risk assessment model were used to investigate the source contributions to PM_2.5 and the human health risks posed by various pollution sources via the inhalation pathway. Almost 50% of the PM_2.5 samples exceeded the secondary standard of China’s air quality concentration limit (75 µg/m³, 24 h). Water-soluble inorganic ions were the main component of PM_2.5 in Zibo, accounting for 50 ± 8% and 56 ± 11% of PM_2.5 at the urban and suburban sites, respectively. OC and OC/EC decreased significantly in the past few years due to enhanced energy restructuring. Pearson correlation analysis showed that traffic emissions were the main source of heavy metals. The Cr(VI) concentrations were 1.53 and 1.92 ng/m³ for urban and suburban sites, respectively, exceeding the national ambient air quality standards limit of 0.025 ng/m³. Secondary inorganic aerosols, traffic emissions, and secondary organic aerosols were the dominant contributors to PM_2.5 in Zibo, with the total contributions from these three sources accounting for approximately 80% of PM_2.5 and the remaining 20% attributed to traffic emissions. The non-carcinogenic risks from crustal dust for children were 2.23 and 1.15 in urban and suburban areas, respectively, exceeding the safe limit of 1.0 in both locations, as was the case for adults in urban areas. Meanwhile, the carcinogenic risks were all below the safe limit, with the non-carcinogenic and carcinogenic risks from traffic emissions being just below the limits. Strict control of precursor emissions, such as SO₂, NOx, and VOCs, is a good way to reduce PM_2.5 pollution resulting from secondary aerosols. Traffic control, limiting or preventing outdoor activities, and wearing masks during haze episodes may be also helpful in reducing PM_2.5 pollution and its non-carcinogenic and carcinogenic health impacts in Zibo.

Polycyclic aromatic hydrocarbons (PAHs) have carcinogenic impacts on human health. However, limited studies are available on the characteristics, sources, and source-specific health risks of PM_2.5-bound PAHs based on personal exposure data, and comparisons of the contributions of indoor and outdoor sources are also lacking. We recruited 101 senior citizens in the winter of 2011 for personal PM_2.5 sample collection. Fourteen PAHs were analyzed, potential sources were apportioned using positive matrix factorization (PMF), and inhalational carcinogenic risks of each source were estimated. Six emission sources were identified, including coal combustion, gasoline emission, diesel emission, biomass burning, cooking, and environmental tobacco smoking (ETS). The contribution to carcinogenic risk of each source occurred in the following sequence: biomass burning > diesel emission > gasoline emission > ETS > coal combustion > cooking. Moreover, the contributions of biomass burning, diesel emission, ETS, and indoor sources (sum of cooking and ETS) to PAH-induced carcinogenic risk were higher than those to the PAH mass concentration, suggesting severe carcinogenic risk per unit contribution. This study revealed the contribution of indoor and outdoor sources to mass concentration and carcinogenic risk of PM_2.5-bound PAHs, which could act as a guide to mitigate the exposure level and risk of PM_2.5-bound PAHs.